Printer Friendly

Myocardial bridging: a rare speciality without clinical significance?/Miyokardiyal kopruleme: Nadir bir ozellik mi?

Myocardial bridges are overall presented in 16% to 80% of the adults due to autopsy based studies (2,3,6). Despite this high prevalence in angiographic studies, incidence of this phenomenon is low (1,2,3,6). This is caused by the fact, that thin bridges cause a little compression which is not seen at angiography. Using provocation tests to enhance systolic myocardial compression (e.g. adenosine) myocardial bridges can be revealed (2,7).

The majority of patients with myocardial bridges are free of any symptoms (1-3). However, myocardial bridging can cause severe complications like atrioventricular blockade, ventricular tachycardia, myocardial ischemia, sudden cardiac death and myocardial infarction (1-7) as shown in the case report by Aytan et al. (8).

Aytan et al. (8) reported the case of a 43-year old patient with non-ST-segment elevation myocardial infarction without coronary artery disease at angiography but a systolic lumen narrowing in the mid proportion of the left anterior descending artery, the typical localisation of myocardial bridges (2,3,6). Authors have clearly shown that a myocardial bridge in this patient caused myocardial infarction possibly by impaired coronary flow reserve and endothelial dysfunction. Aytan et al. treated the patient medically, the "first line" therapy of myocardial bridging (1,2). The patient was followed up to evaluate the success of the B- blocker therapy. To evaluate therapy success and the clinical relevance intravascular ultrasound and intracoronary Doppler ultrasound and pressure devices should be used (1,2,4,6,7).

Patients refractory to medical therapy can be treated by stenting of the tunnelled segment or surgical myotomy and/or coronary artery bypass surgery (1,2,5,6). The risks of surgical and interventional therapy indicate that negative inotropic drugs should be given first.

In conclusion, in younger patients with clinical signs of myocardial ischemia or established ischemia also myocardial bridging should be considered, further data are required to confirm these empirical data.


(1.) Klues HG, Schwarz ER, vom Dahl J, Reffelmann T, Reul H, Potthast K, et al. Disturbed intracoronary hemodynamics in myocardial bridging. Circulation 1997; 96: 2905-13.

(2.) Mohlenkamp S, Hort W, Ge J, Erbel R. Update on myocardial bridging. Circulation 2002; 106: 2616-22.

(3.) Haager PK, Schwarz ER, vom Dahl J, Klues HG, Reffelmann T, Hanrath P. Long term angiographic and clinical follow up in patients with stent implantation for symptomatic myocardial bridging. Heart 2000; 84: 403-8.

(4.) Lovell MJ, Knight CJ: Invasive assessment of myocardial bridges. Heart 2003; 89: 699-700.

(5.) Osula S, Bell GM, Hornung RS. Acute myocardial infarction in young adults: causes and management. Postgrad Med J Heart 2002; 78: 27-30.

(6.) Alegria JR, Herrmann J, Holmes DR, Lerman A, Rihal CS. Myocardial bridging. Eur Heart J 2005; 26:1159-68.

(7.) Escaned J, Cortes J, Flores A, Goicolea J, Alfonso F, Hernandez R, et al. importance of diastolic fractional flow reserve and dobutamine challenge in physiologic assessment of myocardial bridging. J Am Coll Cardiol 2003; 42: 226-33.

(8.) Aytan P, Ulusal G, Coflkun Yenigun E, Yildirim O, Pirpir A, Yildirim S. Muscular bridge causing non- ST-segment elevation myocardial infarction: a case report. Anadolu Kardiyol Derg 2006; 6: 374-5.

Address for Correspondence: Dr. Med. A. Erdogan, Leitender OA Elektrophysiologie Medizinische Klinik I, Kardiologie & Angiologie Universitatsklinikum GieBen, Klinikstr. 36, D-35392 GieBen Tel: 0049-641-9942112 (Pforte)- 0049-641-9942216 (Buro)--0049-641-9942625 (Kath.labor, EPU) Fax: 0049-641-9942229 E-mail:

Ali Erdogan and Harald Greiss Department of Cardiology and Angiology, Justus-Liebig-University of Giessen, Giessen, Germany
COPYRIGHT 2006 Galenos Yayincilik
No portion of this article can be reproduced without the express written permission from the copyright holder.
Copyright 2006 Gale, Cengage Learning. All rights reserved.

Article Details
Printer friendly Cite/link Email Feedback
Title Annotation:Editorial comment/Editoryel Yorum
Author:Erdogan, Ali; Greiss, Harald
Publication:The Anatolian Journal of Cardiology (Anadolu Kardiyoloji Dergisi)
Geographic Code:1USA
Date:Dec 1, 2006
Previous Article:Muscular bridge causing non- ST-segment elevation myocardial infarction/ST-elevasyonsuz miyokard infarktusune neden olan miyokardiyal kopruleme.
Next Article:Coronary rupture to the right ventricle during PTCA for myocardial bridge/"Miyokardiyal Bridge" tedavisinde uygulanan PTKA sirasinda koroner arterin...

Related Articles
Koroner anjiyografi sonrasi gecici korluk/Temporary blindness after coronary angiography.
Mini mi, yoksa kompleks kardiyopleji uygulamasi mi?/Minikardiyopleji yontemi ile kardiyopleji uygulamasi/Which application, complex or mini...
Streptokinaz tedavisiyle iliskili spinal epidural hematom/Spinal epidural hematoma associated with streptokinase therapy.
Myocardial bridge: a bridge to atherosclerosis/Miyokardiyal kopruleme: Ateroskleroza kopru.
Myocardial bridge and atherosclerosis/Miyokardiyal kopruleme ve ateroskleroz.
Cases presented at COPE meeting/COPE toplantisindan vaka bildirimleri.

Terms of use | Privacy policy | Copyright © 2020 Farlex, Inc. | Feedback | For webmasters