Printer Friendly

Man-made chemicals: the hidden role of the lymphatic system in the genesis of polycystic ovary, fibrocystic breast disease, and breast cancer.

Breast cancer, the most frequent maligant disease in women, is caused by synthetic carcinogenic chemicals, or positive ionic poisons (PIPs). Discovered by the late UC Berkeley researcher Albert Krueger, MD, PIPs are synthetic chemicals with a positive ionic charge that are extremely hard to detoxify from the deeper lymphatic system. PIPs can be both carcinogenic and estrogenic and typically throw the female hormone system into chaos, leading to polycystic ovaries and fibrocystic breast disease. They are found in everyday cosmetics, food, air, water, and pharmaceuticals, and leach out of the plastics used to package food, all of which have been strongly linked to carcinogensis.[1-32]


While it is well known that carcinogens fuel carcinogenesis, research is scanty on the mechanisms underlying why and how PIPs are stored in the lymphatic and breast tissue and fail to be properly detoxified and excreted by the majority of today's detoxification protocols. PIPs cripple the deeper lymphatic channels and the biliary networks of the liver, reducing bile production and increasing carcinogenic and estrogenic toxicity. Sadly, research is woefully deficient on PIP toxicity of the deep lymphatic channels in the development of female hormonal disorders and breast cancer.

In the October 1994 Townsend Letter, I stated: "The wellspring of the immunologic defense is the lymphatic system." 33 In the 2001 issue, I started: "We have discovered that a high percentage of IBS sufferers have duodenitis, dysbiosis, stagnant lymph flow, and drainage in the deep lymph channels of the gut. ..."[34] Could the widespread increase in breast disease be somehow related to stagnant lymph flow in the deep lymph channels of the gut? In a 2005 Nature article titled "Unlocking the Drains," we read:
 After centuries of playing second fiddle to the blood system, our
 lymphatic circulation is coming into its own as a key player in
 diseases ranging from cancer to ashthma. Once dismissed as a mere
 drainage network, the body's "second circulation" [lymphatic system]
 is emerging as a crucial player in numerous diseases ... and as a
 vital part of the normal immune system. (35)

All body organs have deep lymphatic drainage functions. Ina alternative medicine, it is critical to consider both deep and superficial channels as integral parts of the lymphatic system. My medical collegues and I have spent over two decades using every sort of manual and electronic lymph drainage device on superficial lymphatic channels, with only minimal success. Since superficial channels converge to form bigger vessels feeding into the thoracic duct or the right lymphatic duct, clinical approaches should be aimed at restoring deep lymphatic drainage as a way to fully cleanse the body of PIPs.

At every level of our physiology, PIPs disrupt our symboises with nature, killing off beneficial commensal cells, so that genetic potential we have for cancer can be played out. The supercintinuum of reciporal and harmonic polarities that entwine us with nature and weave the web of our existence is greatly diminished, because strored lymphatic PIPs cause misalignment that undermines the quality of our movement and flexibility and diminishes our cellular nourishment.

The goal, of quantum medicine is to restore quantic harmonic polarities that are driving force of innate healing. The most important clinical goal in restoring these polarities is to remove PIPs from the body. Quantum medicine views nature as an ally rather than an adversary to be conquered. It honours the instinctive wisdom of the brain's efferent vagus control of the digestive and detoxification process instead of treating the symptoms of maldigestion or commensal cell deficits. When digestion is properly controlled by the autonomic nervous system, commensals become the dominant cells in our anatomy, making it impossible for candida or other opportunistic infections to dominate the gut.

The deep drainage pathways of the abdominal organs drain lymph to the nodes, and situated along it are arteries to preaortic nodes. In every clinical case, the vagus celiac plexus is weak and unable to adequately control the motility and mobility of the digestive, detoxification, and lymphatic systems. When this happens, the body cannot detoxify and excrete PIPs. Thus, they end up impacted in deeper lymph channels, reducing circulation (nourishment) and innervation to deep organs.

Cancer is a complex process that involves acute and chronic cell injury and what researches are calling ion deregulation. Citing over 30 scientific papers, Dr. Benjamin F. Trump and colleagues at the University Of Maryland School of Medicine state: "We propose that ion deregulation is a major mediator in the process and, therefore, provides a critical link between acute cell injury, tumor promotion, and carcinogenesis." (36-46)

PIPs can act as receptors for certain pathogenic microbes causing free floating yeast, fungi, and bacteria to form treatment-resistant biofilms. (46) Large amounts of exopolysaccharide are produced that envelop biofilm, making it extremely resistant to antibiotics and any kind of anti-infective treatment. PIPs disrupt the network of ion channels and thus can impregnate deep lymphatic channels. After over a decade of kinesiological and digital infrared thermographic evidence in these patients, it is common to find a diminishment of the greater splanchnic nerve/celiac plexus with stubborn spinal fixations around the vertebrae T8 and C7. (47-49).

Detoxification of PIPs in the Deep Lymphatic Networks

PIPs cause oxidative stress and abnormalities of immune TH-1/TH-2 mediated immune responses with resultant ion deregulation. The first step in releasing these toxins is to restore the function of commensal microflora. These living cells exceed the number of human cells by a factor of 10. When plentiful, commensals manufacture powerful detoxifying compounds with a strong polarity that makes it possible to dislodge PIPs from the deeper lymphatic networks and tissues of the body. The second step is to find effective ways to increase the production of bile so that they are safely eliminated from the body, as I have explained in greater detail in previous columns. (53)

Comprising over 90% of the body's cells, commensals produce a goldmine of anticancer agents and detoxifying compounds that are part of the body's immunological weaponry. (50-57) When fed synbiotics, these amazing cells can quickly trigger and magnify the immune response and amplify immune mucosal mechanisms. However, our long-term clinical experience reveals that the majority of today's probiotic products lack the synbiotic counterparts to restore deficient commensal cells and are not complete microflora-balancing products. To find a permanent residence in the gut, they have to be fermented in stages in an appropriate noncompetitive microbe-to-microbe balance and nourished with predigested and fermented synbiotics. In this manner, they can successively thrive and flourish in the body and express their rich genetic diversity in ways that will take researchers decades to fully understand. Since commensal cells are vast reservoirs of essential hard-to-get nutrients and anti-inflammatory compounds that soothe inflammation more effectively than any anti-inflammatory pharmaceutical or nutraceutical, it is essential that we find innovative ways to rapidly feed, cleanse, and fortify them. My long-term research on synbiotics has been independently substantiated by doctors in cases of severe acute pancreatitis, chronic hepatitis, abdominal surgery, liver transplantation, and abnormal calcification. (54-57)

Most detoxification methods are a double-edged sword that have the potential to amplify malignancies. (51-53), (61-64) By focusing on immunorestoration instead of immuno-modulation, we can cleanse the body more efficiently and remove the harmful xenoestrogens that cause polycystic ovary and fibrocystic breast disease. Altough the changes in breast tissue that a woman experiences when she has fibrocystic breast disease are benign, this occurrence most commonly marks the beginning of a deep lymphatic deficit and excessive storage of PIPs that are xenoestrogens. (58) And, women living in an urban area of an agricultural and industrial region of Argentina had high levels of organochlorine residues in their breast adipose tissue invasive breast carcinoma. (59) Indeed, epidemiologic studies have identified tumor size, and cell proliferation with lymph node involvement. (60)

In summary, the Canadian Breast Cancer Network stated: "Women are told by the media, by society, and by the government that the situation of breast cancer is on the mend. ... The basic fact remains that the medical community cannot defintely identify the causes of cancer, nor can they 'cure' cancer." Clearly, it is a deferral of normal and natural innate immune reciprocity that induces an ever-widening, self-propagating wave of tissue destruction that underlies chaotic female hormone responses and carcinogenesis. Restoring healthy and powerful immune responses via commensal cells can manintain optimal rebalancing of reciprocal TH-1/TH-2 responses and give the innate immune system its best shot at conquering cancer and quelling the inflammation that underlies the hormonal derangements plaguing women.

The normal immune response is in constant reciprocal motion, and its repertoire is straightforward: optimal synbiotic and probiotic rebalancing of reciprocal TH-1 and TH-2 responses determines one's level of health and the power of the immune response against carcinogenesis. Rather than using fragmented, single-nutrient, or anti-infective approaches, addressing the abberant core physiological issues underlying carcinogenesis will yield the best clinical outcome. Finally, nature's recipes for nourishment and detoxifcation activate the full operational complexity of the immune system and can remove us from the realm of performing unethical and potentially lethal oncological or other medical treatments.

The author is supported in part by American Academy of Quantum Medicine, a nonprofit foundation dedicated to frontier research in quantum medicine. The author is affiliated with QuantaFoods LLC, a firm that develops and researches probiotics and fermentation to develop novel form of synbiotic nutrients, and is a research consultant of several independent, university-based laboratories. The content of this article was neither influenced nor constrained by these facts.


(1.) Henderson BE et al. Hormonal Chemoprevention of cancer in women. Science. 1993 Jan 29;59(5095):633-638.

(2.) Feuer EJ, Wun LM. How much of the recent rise in breast cancer incidence can be explained by increases in mammography utilization? A dynamic population model approach. Am J Epidemiol. 1992 Dec 15; 136(12):1423-1436.

(3.) Hahn RA et al. Nulliparity, decade of first birth, and breast cancer in Connecticut cohorts, 1855 to 1945: an ecological study. Am J Public Health. 1989 Nov; 79(11): 1503-1507.

(4.) Hoel DG et al. Trends in cancer mortality in 15 industrialized countries, 1969-1986. J Nati Cancer Inst. 1992 Mar 4;84(5):313-320.

(5.) Kohlmeier L et al. Lifestyle and trends in worldwide breast cancer rates. Ann N Y Acad Sci. 1990;09:259-268.

(6.) Henderson BE et al. Toward the primary prevention of cancer. Science. 1991 Nov 22;254(5035):1131-1138.

(7.) Westin JB et al. The Israeli breast-cancer anomaly. Ann N Y Acad Sci. 1990;609:269-279.

(8.) Fishman J et al. Effect of malnutrition on the metabolism of sex hormones in man. Clin Pharmacol Ther. 1977:721-728.

(9.) Porter WP et al. Toxicant-disease-environment. Science. 1984 Jun 1;224(4652):1014-1017.

(10.) Bartley J et al. Metabolism of benzo(a) pyrene by human epithelial and fibroblastic cells: metabolite patterns and DNA adduct formation. J Cell Biochem. 1982;18(2):135-148.

(11.) Ekbom A et al; Evidence of prenatal influences on breast cancer risk. Lancet. 1922 Oct 24;340(8826):1015-1018.

(12.) Hsieh CC et al. Twin membership and breast cancer risk. Am J Epidemiol. 1992 Dec 1:136(11):1321-1326.

(13.) Trichopoulos D et al. Urine estrogens and breast cancer risk factors among postmenopausal women. Int J Cancer. 1987 Dec 15;40(6):721-725.

(14.) Manz a et al. Cancer mortality among workers in chemical plant contaminated with dioxin. Lancet. 1991 Oct 19;338(8773):959-964.

(15.) Kurastsune M et al. A cohort study on mortality of "yusho" patients: a preliminary report. Preincess Takamatsu Symp. 1987;18:61-66.

(16.) Bertazzi PA et al. Ten-year mortality study of the population involved in the Seveso incident in 1976. Am J Epidemiol. 1989 Jun; 129(6): 1187-1200.

(17.) Dusich K et al. Cancer rates in a community exposed to low levels of creosote components in municipal water. Minn Med. 1980 Nov;63(11):803-806.

(18.) Dean AG et al. Adjusting morbidity ratios in two communities using risk factor prevalence in cases. Am J Epidemiol. 1988 Mae;127(3):654-662.

(19.) Hall NE; Rosenman, KD. Cancer by industry: analysis of a population-based cancer registry with an emphasis on blue-collar workers. Am J Ind Med. 1991;19(2):145-159.

(20.) Griffith J et al. Cancer mortality in U. S. counties with hazardous waste sites and ground water pollution. Arch Environ Health. 1989 44(2):69-74.

(21.) Unger M et al. Organochlorine compounds in human breast fat from deceased twith and without breast cancer and in biopsy material from newly diagnosed patients undergoing breast surgery. Environ Res. 1984 Jun;34(1):24-28.

(22.) Falck F Jr et al. Pesticides and polychlorinated biphenyl residues in human breast lipids and their relation to breast cancer. Arch Environ Health. 1992 47(2):143-146.

(23.) Mussalo-Rauhamaa, H et al. Occurrence of beta-hexachlorocyclohexane in breast cancer patients. Cancer. 1990 Nov 15;66(10):2124-2128.

(24.) Wolff MS et al. Blood levels of organochlorine residues and risk of breast cancer. J Natl Cancer Inst. 1993 Apr 21; 85(8):648-652.

(25.) Donna A et al. Ovarian mesothelial tumors and herbicides: a case-control study. Carcinogenesis. 1984 Jul;5(7):941-942.

(26.) Donna A et al. Triazine herbicides and ovarian epithelial neoplasms. Scand J Work Environ Health. 1989 Feb; 15(1)47-53.

(27.) Claus EB et al. Genetic analysis of breast cancer in the cancer and steroid hormone study. Am J Hum Genet. 1991 Feb; 48(2):232-242.

(28.) Welch RM et al. Estrogenic action of DDT and its analogs. Toxicol Appl Pharmaco. 1969 Mar;14(2):358-367.

(29.) Coosen R; van Velsen, FL. Effects of the beta-isomer of hexachlorocyclohexane on estrogen-sensitive human mammary tumor cells. Toxicol Appl Pharmacol. 1989 Nov; 101(2):310-318.

(30.) Maltoni C et al. Benzene, an experimental multipotential carcinogen: results of the long-term bioassays performed at the Bologna Institute of Oncology. Environ Health Perspect. 1989 Jul; 82:109-124.

(31.) Huggins C et al. Induction and extinction of mammary cancer. A striking effects of hydrocarbons permits analysis of mechanisms of causes and cure of breast cancer. Science. 1962 Jul 27;137:257-262.

(32.) Wassermann M et al. Organochlorine compounds in neoplastic and adjacent apparently normal breast tissue. Bull Environ Contam Toxicol. 1976 Apr; 15(4):478-484.

(33.) Yanick P. Functional disturbances of the liver and lymphatic system. Townsend Letter. 1994 Oct.

(34.) Yanick P. Food and supplement benefits and risks in catcinogenesis--II. Townsend Letter. 2001 Nov.

(35.) Brown P: Unblocking the drains. Nature. 2005;436:28.

(36.) Trump BF et al. Sodium and calcium regulation and the role of the cytoskeleton in the pathogenesis of disease: a review and hypothesis. 1981. Scan Electr Microsc. 2:435-454.

(37.) Trump BF et al. lon regulation and the cytoskeleton in preneoplastic and neoplastic cells. In: Harris CC, Autrup H, eds. Human Carcinogenesis. New York: Academic Press; 1983. 35-84.

(38.) Trump BF et al. The role of calcium in cell injury and repair: a hypothesis. 1985 Surv syn Path Res. 4:248-256.

(39.) Trump BF et al. The role of ion regulation in respiratory carciongenesis. In: McDowell EM, ed. Lung Carciomas (Current Problems in Tumour Pathology). Edinbugh: Churchill Livingstone; 1987:162-174.

(40.) Trump BF et al. Carcinogenesis. 1987;8(8):1027-1031.

(41.) Teoh D et al. Giardia lamblia rearranges F-actin and a-actinin in human colonic and duodenal monolayers decreases transepithelia electrical resistance. J Parasitoloy. 86(4):800-806.

(42). Morck DW et al. Microbial biofilms: prevention, control, removal. In: Seymour S, ed. Disinfection, Sterilization, and Preservation. 5th ed. Lippincott Williams & Wilikins: Baltimore, MD; 2004:675-683.

(43.) Ceri H et al. The Calgary Biofilm Device: Measurement of antimicrobial sensitivity of bacterial biofilms. J Clin Microbiol. 1999;37:1771-1776.

(44.) Prendergast MM et al. In vivo phase variation and serological response to lipooligosaccharide of Campylobacter jejuni in experimental human infection. Infection and Immunity. 2004;72:916-922.

(45.) Knaipes Ml et al. A deep-rough matanl Campylobacter jejuni non-invasive, intestinal epithelial cells. Infection & Immunity. 2004;72:2452-2455.

(46.) Moran AP et al. Recent advances in understanding biofilms of mucosae. Reviews Enviro Science & Biotechnology. 2003;2:121-140.

(47.) Harrell JC et al. Estrogen receptor positive breast cancer metastasis: altered hormonal sensitivity and tumor aggressiveness in lymphatic vessels and lymph nodes. Cancer Res. 2006;66:9308-9315.

(48.) Harrell JC et al. Estrogen insensitivity in a model of estrogen receptor positive breast cancer lymph node metastasis. Cancer Res. 2007;67:1058-10591.

(49.) Harrell JC et al. Metastasis signatures changes completely when whole-tissue compared to laser capture microdissection are used for gene expression profiling: breast cancers versus lymph node metastases. Clin Exp Metastasis. 2007 Oct.

(50.) Bengmark S. 2001 Curr Opin Clin Nutr Metab Care. 5)6).

(51.) Yanick P. Strengthening the innate ability of the immune system to fight cancer by restoring thymic and TH-1 and TH-2 cytokine responses. Townsend Letter. 2006 Aug-Sept.

(52.) Yanick P. Restoring immunity, the autonomic nervous system, and bioenergetic reciprocity for a fuller expression of innate healing in naturopathic medicine Townsend Letter. 2007 Apr.

(53.) Yanick P. Strengthening innate immunological weaponry against cacer. Townsend Letter. 2007 Aug-Sept.

(54.) Bengmark S. Use of pro- pre- and synbiotics in the ICU--future options, In: Shikora SA, Martindale RG, Schwaitzberg SD, eds. Nutritional Considerations in the Intensive Care Unit--Science, Rationale and Practice. Dubuque, IA: Kendall/Hunt Publishing Company; 2002:381-399

(55.) Bengmark S. Gut microbial ecology in critical illness: is three a role for pre- pro- and synbiotics. Curr Opin Crit Care. 2002;8:145-151.

(56.) Bengmark S. Aggressive peri- and intraoperative enteral nutrition--strategy for the future. In: Shikora SA, Martindale RG, Schwaitzberg SD, eds. Nutritional Considerations in the Intensive Company; 2002:365-380.

(57.) Bengmark, S, 2001 pre- pro-, and synbiotics. Curr Opin Clin Nutr Metab Care 2001;4(6): 571-579.

(58.) D'Amelio R et al. Association between PCO and fibrocystic breast disease. Gynecol Obstet Invest. 2001; 134-37.

(59.) Munoz-de-Toro M et al. Organochlorine levels i adipose tissue of women from a littoral region of Argentina. Environ Res. 2006,102:107-112.

(60.) Haues DF et al. Prognostic factors in breast cancer: current and new predictors of metastasis. J Mammary Gland Biol Neoplasia. 2001;6:375-392

(61.)Paolini M, Nestle M. Pitfalls of enzyme-based molecular anticancer dietary manipulations: food for thought. Mutat Res. 2003;181-189.

(62.) Guidelines on diet, nutrition and cancer prevention: reducing the risk of cancer with healthy food choices and physical activity. The American Cancer Society Advisory Committee on Diet, Nutrition and Cancer Prevention. Ca Cancer J Clin. 1996;325-341.

(63.) Paolini M. Brussels sprouts: exceptionally rich sources of ambiguity in anticancer strategies. Toxocol Appl Pharmacol. 1998:293-294.

(64.) Paolini M. Nature. 1992;357:48.

by Paul Yanick, Jr., PhD
COPYRIGHT 2009 The Townsend Letter Group
No portion of this article can be reproduced without the express written permission from the copyright holder.
Copyright 2009 Gale, Cengage Learning. All rights reserved.

Article Details
Printer friendly Cite/link Email Feedback
Title Annotation:Quantum Medicine Update
Author:Yanick, Paul, Jr.
Publication:Townsend Letter
Geographic Code:1USA
Date:Apr 1, 2009
Previous Article:Natural treatment of osteoporosis and breast disease and the recurring epidemic of iodine deficiency.
Next Article:A suggested pattern discrimination for premature ovarian failure (POF).

Related Articles
Finally--a test for iodine sufficiency. (Thyroid Update!).
Why wearing a bra can be hazardous to your health.
Commentary: breast cancer's environmental legacy: how every day toxins threaten women's health.
Risk factors for male breast cancer.
Diet and supplements for benign breast disease.

Terms of use | Privacy policy | Copyright © 2018 Farlex, Inc. | Feedback | For webmasters