The metabolic syndrome; epidemiology, clinical treatment, and underlying mechanisms.9781588297389 The metabolic syndrome metabolic syndrome n. See syndrome X. Metabolic syndrome A group of risk factors for heart disease, diabetes, and stroke. ; epidemiology, clinical treatment, and underlying mechanisms. Ed. by Barbara Caleen Hansen and George A. Bray. Humana Press Inc. 2008 401 pages $149.00 Hardcover Contemporary endocrinlogy RC662 As we reach a better understanding of the need for early intervention and prevention to slow or halt Metabolic Syndrome, we also need to understand the basic science to develop better treatment. With the goal of stimulating new research and better therapeutics, contributors from epidemiology, physiology, molecular biology, molecular biochemistry and clinical practice offer cross-disciplinary analyses on this complex disease. Topics here include the role of the rise of obesity in insulin resistance, treatment options (weight loss, exercise, hormones and surgery), links with cardiovascular disease from an epidemiological perspective, influence of the sympatho-adrenal system, insulin action and endothelial endothelial /en·do·the·li·al/ (-the´le-al) pertaining to or made up of endothelium. Endothelial A layer of cells that lines the inside of certain body cavities, for example, blood vessels. function, vascular disease in pre-diabetic animal models, highly sensitive C-reactive protein, insulin signaling, insulin resistance and dyslipidemia, pancreatic islet islet /is·let/ (-lit) an island. islets of Langerhans irregular microscopic structures scattered throughout the pancreas and comprising its endocrine portion. therapy in obesity; influence from glucagon glucagon (gl  `kəgŏn), hormone secreted by the α cells of the islets of Langerhans, specific groups of cells in the pancreas. It tends to counteract the action of insulin, i.e. peptides, insulin receptor
substrates, tyrosine kinase, muscle fat, alterations in atypical protein
kinase C Protein kinase C ('PKC', EC 2.7.11.13) is a family of protein kinases consisting of ~10 isozymes.[1] They are divided into three subfamilies: conventional (or classical), novel, and atypical based on their second messenger requirements. activation, the liver and glucose homeostasis homeostasisAny self-regulating process by which a biological or mechanical system maintains stability while adjusting to changing conditions. Systems in dynamic equilibrium reach a balance in which internal change continuously compensates for external change in a feedback ; and the chronomics of the syndrome. ([c]20082005 Book News, Inc., Portland, OR) |
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