Diabetes Keys via God's Blueprints, Has Nutrient Inhibitors Disabled the Metabolism Insulin Trigger...

This is a Hypothesis on the disruptions seen in Diabetes and examples of how they could be connected to the excessive consumption of Nutrient Inhibitors such as Oxalic and Phytic Acids and Phytoestrogens found in the processed food-chain, causing "Nutrient Starvation" leading to "Cell Starvation".. These connections are arrived at by following God's Blueprints, which are the Internal Systems the body was created with.. These examples show why I am absolutely convinced the Culprit behind Diabetes and the Root Cause of the Auto-Immune Disease Epidemic is the excessive consumption of Nutrient Inhibitors resulting in "Nutrient Starvation" causing "Cell Starvation" resulting in the silient killer, a prolonged "Internal Stress Response"... This Hypothesis maps out how Diabetes is the symptoms manifested when "Cell Starvation" is present, and backed-up by using God's Blueprints to prove it.. When these Blueprints are followed the cause of all the Auto-Immune diseases are perceivable.. And diseases such as Alzheimer's, Autism, Heart Disease, High Blood Pressure, Cancer, Diabetes, Parkinson's, Birth Defects, Multiple Sclerosis (MS), MG, Multiple Dystrophy (MD), Aids, Peripheral Nerve Disorders, Lupus, Scleroderma, Epilepsy, Blood Disorders, Rare Diseases, etc. could really be cured if we all get on the same page and start working with God's Internal Systems instead of against them..

I wonder if it is Nutrient Inhibitors disabling the Insulin ON-OFF Switch in the Metabolism Process..

I am not a doctor and this Hypothesis contains absolutely no medical advice.. It is just my personal attempt to show how the disruptions in Diabetes could be stemming from exposure to excessive amounts of nutrient inhibitors from the processed food chain, which could lock Insulin out of the Metabolic process....

List of Symptoms and Disorders associated with Diabetes:
#High blood Glucose levels (Hyperglycemia)..
#In Type I Diabetes Mellitus destruction of Pancreatic Beta Cells, with cellular metabolism similar to starving...
#Type II Diabetics: Excessive Insulin but Target cells become desensitized to it due to a connection to down-regulation of Insulin receptors problems..
#Excessive thirst, hunger and frequent, excessive urination due to the inability to reabsorb water...
#Dysfunction in the Posterior Pituitary Gland...

Diabetes,I believe,is due to the excessive consumption of Nutrient Inhibitors (such as Oxalic and Phytic Acids and Phytoestrogens that are reported to be found in Soy)in the processed food chain that is blocking the body's ability to utilize nutrients,such as Iron and Calcium... By creating insoluble Iron complexes and Insoluble Calcium Salts this creates an electrolyte imbalance, which denatures functional proteins and disrupts Hemoglobin's ability to transport Oxygen... This would create a deficiency of Oxygen in the Cellular Respiration Electron Transport Chain, which would in turn disrupt the metabolism of ATPs... Since Glucose is the preferred fuel for making ATPs, insufficient ATPs would cause the Internal Negative Feedback System for Regulating Glucagons /Insulin to react in an attempt to restore the depleted ATP resources.. Thus a Hypoglycemia response would be triggered and locked in..(So when faced with a problem concerning the Insulin On-Off Switch,I believe the place to start looking would be in the Glucagons/Insulin Regulation Negative Feedback System.. That is where the Glucagon and Insulin switch mechanisms are yoked together.. And this yoked switch mechanism is also tied to the Metabolism process)..

My opinion concerning Diabetes is : Hypoglycemia is the Internal Systems' interpretation of the problem and Hyperglycemia is the adjustment to get to Homeostasis and this Hyperglycemia adjustment is the medical communities' interpretation of the problem..

Hypoglycemia:
Hypoglycemia is insufficient blood glucose levels which results in increases secretion of Epinephrine, hGH and Glucagon.. This causes weakness, sweating, tremor, anxiety, hunger and increased heart rate...(So when the Internal Systems detect that the ATPs are deficient, their response would be to gather fuel to make the ATPs, so they would instantly summons Glucose to accumulate in the blood to serve as the ATP making fuel.. But since the ATP metabolism problem has been brought on by the Nutrient Inhibitors' effect on Iron and Hemoglobin, which disrupts the delivery of Oxygen to the metabolism process, the accumulation of Glucose would be to no avail,for Glucose is not the culprit... This would only serve to make Glucose blood levels excessive)... As ATPs continues to diminish then the Brain Cells begin to lose their ability to carry out their physiological functions.. (In a methodical attempt to bring the body back out of insufficient nutrient "Shock Stress", the General Adaptation Syndrome Stress Response would be to keep resetting the threshold higher for the Glucose levels in the blood.. At first short termed "Fight or Flight" Stress Responses would be activated and then replaced with hormonal driven long-term reactions in the Resistance Reaction Stress Response.. Epinephrine and hGH would be released in a "Shock Stress" response.. And the Glucagon would be released if the body perceived the functions that required Glucose were lacking.).

The dominant controlling hormones, in the "Resistance Reaction" Stress Response would beCorticotropin Releasing Hormones (CRH), Thyrotropin Releasing Hormone (TRH) and the Growth Hormone Releasing Hormone (GHRH)... This unites the actions of Cortisol, TSH and hGH, which functions to produce ATP for the chemical activity in the cells, by increases the destructive metabolism (Catabolism) of Proteins, Non-carbohydrates, Triglycerides and depleting the Glycogen stores to make Glucose to serve as fuel for making ATPs...

CRH stimulates secretion of (ACTH) which stimulates secretion of Cortisol... Cortisol has many influences: Cortisol increases blood pressure, by making blood vessels more susceptible to constriction stimuli... Cortisol decreases inflammation in order to keep it from becoming disruptive but in the process removes its protective qualities.. Cortisol inhibits development of new connective tissue,thus causing wound healing to slow down... Cortisol increases the destructive metabolism (Catabolism)of Proteins... Cortisol triggers(Gluconeogenesis) which is the conversion of non-carbohydrates into Glucose..

TRH stimulates the secretion of (TSH) Thyroid-Stimulating Hormone, which stimulates secretion of T3 and T4, Thyroid Hormones... These Hormones are the ones responsible for stimulating the formation of ATP from Glucose..

GHRH stimulates the secretion of Human Growth Hormone (hGH)... Long-term release of hGH can be a major culprit in Diabetes.. hGH activates the (Glycogenolysis) which is the conversion of Glycogen into Glucose... hGH also activates the destructive metabolism of Triglycerides...

In the Resistance Reaction Stress Response Aldosterone secretion increases... Aldosterone's role in the Stress Response,is to fend off Acidosis and maintain the body pH by conserving Na+ and eliminating the excessive Hydrogen Ions that builds-up when destructive metabolism dominates... The role of Na+ is to maintain a high Blood Pressure in the Fight or Flight Stress Response.. Na+ leads to water retention.. The Resistance Reaction Stress Response Phase lets the body continue fighting the altered blood chemistry, which is the "Stressor", and the goal is to provide sufficient ATP and enzymes and alter the circulatory system to resist this threat and return the blood chemistry back to normal.. If this blood chemistry is returned to normal then the cells would be able to use Glucose at the same rate it is entering into the blood and this would allow the reset mechanism in the General Adaptation Syndrome to return to normal and blood glucose levels would become normal... If the blood chemistry is not corrected and the cells ability to get nutritional rich blood remains limited then the General Adaptation Syndrome goes into a State of Exhaustion.. If the Stressor remains for too long a period of time, then the battle can be lost due to pure exhaustion, because the body's hormone, resources and other mechanisms become depleted and start failing.. (So just suppressing symptoms, dooms the body to failure, it would seem. This may account for there being no cures.)..

Hyperglycemia:
Glucose is the preferred fuel for making ATP.. In Homeostasis maintaining ATP levels, which is cell energy, takes president over everything else..(For this reason,I believe the Culprit behind the Diabetics Hyperglycemia is insufficient ATP Metabolism... I believe that the original Diabetes problem is an Internal perception of Low Glucose due to deficient ATP metabolism caused by the lack of Oxygen brought on by the excessive consumption of the Nutrient Inhibitors which created Insoluble Iron Complexes and Insoluble Calcium Salts which altered the Electrolyte concentration creating an imbalance which created a hostile internal environment which denatures the functional protein Hemoglobin, and disrupts its ability to carry Oxygen, which in turn disrupted the Cellular Respiration Electron Transport Chain's metabolism of ATPs.. And this would cause the Internal Systems to react as though the body had Hypoglycemia or low blood Glucose, this would activate the General Adaptation Syndrome(GAS)in a Stress Response and this would reset the internal control of the threshold level of blood Glucose to a higher degree.. And this new higher threshold level, when finally discovered by the medical community, would be seen as abnormally high and would be perceived as the culprit... But in my opinion, this higher than normal threshold for blood glucose level is only an adjustment made by the Internal Systems in an attempt to restore ATP homeostasis)...

Acidosis and Functional Protein Disruption:
Functional Proteins perform many essential functions: Antibodies/ Hormones/ Transport Proteins/ Contraction Proteins/ Enzymes... When they are denatured they cannot perform their physiological roles, because their important chemical interacting sites are destroyed.. A low pH, high acidity state enhances this denaturing... Hemoglobin is a transport protein, and when exposed to an acidic environment it completely loses the ability to bind Oxygen for transportation... The acidic environment destroys the hemoglobin structure disrupting its ability to carry the oxygen... Also effected by this denaturing are the acid-derived, protein and peptide hormones involved in metabolic regulation, development and growth, such as Insulin and growth hormone (gH)... (So this denaturing of functional proteins begins with the Nutrient Inhibitors causing an altered concentration of usable Iron and Calcium electrolytes, which causes a hostile environment which denatures proteins... As the proteins become denatured and hemoglobin cannot transport sufficient Oxygen to the Cellular Respiration Electron Transport Chain, thus inhibiting production of ATPs, this enhances acidity which creates an even more hostile environment, fast-tracking the denaturing of proteins.)...

Negative Feedback Regulation of Glucagon and Insulin:
Glucagon's primary role is to increase blood glucose levels... Insulin's primary role is to lower blood glucose levels..
The following is a scenario of circumstances that would take place in a one sided disruption of the Glucagons/Insulin Regulating Negative Feedback System:
(These circumstances are based on my theory that Insulin has been deactivated by the "Internal Systems" or desensitized its effects in the metabolism process... And deficient ATP would work both the Glucose angle and the Locked-down Insulin angle by activating the ON-OFF switch mechanisms in the Negative Feedback Regulation of Glucagon and Insulin.. This is a situation the excessive consumption of Nutrient Inhibitors could create.).

Insufficient Glucose Response:
#1.The Alpha Cells release Glucagon when the blood is low in glucose,(I believe insufficient ATP metabolism would trigger a need for Glucose and lock in this mode until the ATP metabolism becomes sufficient.)...
#2.The Glucagon stimulates the liver cells to convert the stores of Glycogen to Glucose (Glycogenolysis) and to change Lactic Acid and some Amino Acids into Glucose (Gluconeogenesis), in order to raise blood glucose levels..
#3.Normally this would bring Homeostasis and everything would settle down.. The higher blood level of Glucose should inhibit the release of Glucagon and stimulate the release of Insulin by the beta cells..(But with the Nutrient Inhibitors blocking Oxygen and disrupting the metabolism of ATP, the "Fight or Flight" Stress Response from the Sympathetic Division of the Autonomic Nervous System over-rides the normal by resetting the internal environment controls in the General Adaptation Syndrome(GAS),thus raising the Glucose threshold level... So in my opinion, the Insulin side of the Negative Feedback Regulation will remain turned-off, until the Nutrient Inhibitor problem is corrected and Homeostasis is restored.)...
In which case, Insulin will not be available to:
#1. Speed Glucose entrance into the cells..
#2. Speed up the conversion of Glucose into Glycogen, so it can be stored...
#3. Increase the absorption of Amino Acids...
#4. Increase the building-up of Proteins...
#5. Inhibit the conversion of Glycogen into Glucose..
#6. Inhibit the conversion of Lactic Acid and certain Amino Acids into Glucose..
#7. Increase the building-up of Fatty Acids (Lipogenesis)..

These effects on the metabolism process would account for the fact that the cellular metabolism in a type I diabetic, if left untreated is similar to that a person that is starving...(Because,in my opinion,this is exactly what us taking place, the Nutrient Inhibitor are causing "Cell Starvation".. The effects of this disruption of the Neg. Feedback System would cause: Deficient reserves of Fatty Acids, Proteins, Amino Acids, Glycogen, Cell Glucose and Insulin and disrupt all the processes they are involved in... Plus there would be excessive use of Lactic Acid and Amino Acids as a fuel for making Glucose disrupting these resources for other uses... There would also be excessive Glucagon and excessive blood levels of Glucose...(In my opinion the outlet for getting rid of the excess Glucose is locked down due to the inability to utilize it, because of the deficient Oxygen in the Cellular Respiration Chain)..

Several Health Disruptions Stemming from the Negative Feedback Disruption:

If a cell is unable to retain Glucose, it will turn to destructive metabolism (catabolism) of Fatty Acids for making ATP (cellular energy).. This type of metabolism of fatty acids proteins and Triglycerides causes weight loss and Ketoacidosis, disrupting the pH...(In my opinion this would enhance the Acidosis that began with the disrupted ATP metabolism.).. As the constituents of these fats(lipids) are transported through the blood some of the lipid particles get deposited on the blood vessel walls causing arterial disease (Atherosclerosis)...Which branches into many Cardiovascular Disruptions, such as deficient blood flow to the brain (Cerebrovascular Deficiency), insufficient blood flow to the heart(Ischemic), deficient blood causing tissue death (Gangrene) and Peripheral Blood vessels disruptions..

Excessive blood Glucose causes eye problems: Retina blood vessel damage, Cataracts due to excessive Glucose adhering to the proteins of the lens causing cloudiness.. Excessive Glucose also injures the renal blood vessels, damaging the Kidneys...

Dysfunctional Posterior Pituitary Gland:
The Posterior Pituitary Gland has specialized Neuroglia Cells called Pituicytes... (When Lysosomes are deficient of Oxygen they become fragile rupture and burst releasing their nerve cell destroying enzymes... These enzymes destroy Neurons and Neuroglia.. This is a possible reason for a dysfunctional Posterior Pituitary Gland, which could be attributed to the Nutrient Inhibitors disruption in the transportation of Oxygen.)...

Excessive Thirst, Urination and Eating and even Disorientation:
(These disruption brings us back to the disrupted ATP Metabolism and prolonged secretion of the Hormones in the Resistance Reaction Stress Response and Cell Starvation of Nutrients.)..

One method, used by the medical community, for breaching the Blood-Brain Barrier is to inject a person with a concentrated sugar solution... The Osmotic effect caused by the excessive sugar concentrate causes the Endothelial Cells to shrink and pull apart which allows gaps to open up in the region between the cell membranes which normally serve as a selective barrier for material passage..(In my opinion this same, or a very similar effect could be taking place with the prolonged secretion of hGH in the Resistance Reaction Stress Response and the disrupted Insulin Switch in the Neg. Feedback Reg. of Glucagon and Insulin and the reset threshold for the Glucose levels.)..

Pro-longed excessive secretion of hGH has a "Lack of Insulin" effect on diabetes.... Excessive hGH causes high blood Glucose concentration.. Continuous Hyperglycemia stimulates the continuous secretion of Insulin... If this continues for a prolonged period of time this can lead to "Beta-Cell Burnout"... This limit's the pancreatic beta cells' ability to synthesize and secret Insulin.. (So, it just stands to reason, an "Insufficient Nutrient Stress Response" with the prolonged release of hGH could result in a "Beta Cell Burnout" or lead to a desensitized state which would mean the (GAS) has reset the threshold for Glucose levels, thus causing excessive high concentrations of Glucose (sugar) in the blood, which would cause a disruption in the Osmotic effect that could breach the Endothelial Cells' selective barrier, in the Brain...
(I believe else where also, such as the Endothelia-Capsular Membrane)... Normally the Endothelia-Capsular Membrane's basement membrane and slit membranes control passage of nutrients and permits less than 1% of the main plasma protein to escape through the filter, but Blood Pressure increases this filtration... If blood pressure gets high enough it can force the larger plasma proteins and formed elements through... (So combining the higher Blood Pressure in a Stress Response and the higher sugar concentration in the blood, it is quiet possible the Endothelial-Capsular Membrane has been made more permeable).. If the Endothelial-Capsular Membrane have been made more permeable this would disrupt filtration, and this could lead to the Nephrotic Syndrome, which in simplified terms is protein in the urine aka protein-uria... This increased permeability would allow Albumin and other proteins to escape from the blood and be lost in the Urine, depleting their resources.. Albumin loss from the blood lowers Blood Colloid Osmotic Pressure and this causes water to leave the blood and flow into the Interstitial Spaces.. Edema is a result of this disruption.. Another disruption from the loss of Blood Protein is Hyperlipidema in simplified terms this means blood levels of Phospholipids, Triglycerides and Cholesterol are high.. One of the causes of Nephrotic Syndrome is diabetes mellitus...

Glucosuria is the situation in which the blood concentration of glucose rises above the level at which the Na+-Glucose secondary active transporters can reclaim it, thus surpassing the threshold of the Transport maxium(?).. The most common cause of Glucosuria is diabetes mellitus.. The results of Glucosuria is the excess glucose spills over into the urine the causes an increase in urine... Reason for this is the glucose is not reabsorbed disrupting the osmosis effect, so water molecules are also left behind causing excessive urine.. (Both Glucosuria and Nephrotic Syndrome have been attributed to being caused by Diabetes... I think they are all present and are all three just symptoms of the disruption in the ability to absorb nutrients caused by the excessive consumption of Nutrient Inhibitors. That is why I believe the medical communities very first line of treatment for people with diseases should be a very thorough, absolute, complete, trustworthy Cell Nutrient Test, not just a run of the mill blood test.. It seems to me, the run of the mill test would be misleading because the nutrients would be there but the cells are unable to utilize them... This is the answer we need to know.)..

Aldosterone effect on Water Dehydration :
Aldosterone is released in the Stress Response, Low Blood Pressure Negative Feedback Response and the Shock Response: all are "Internal Systems" which would possibly be triggered by the "Stressor" of insufficient nutrients, which could be brought on by the excess consumption of Nutrient Inhibitors in the processed food chain... Aldosterone, along with the (ANP) Atrial Natriuretic Peptide and (ADH) the antidiuretic hormone are the regulators of the level of sodium (Na+) in the blood... Na+ plays a major role in maintaining electrolytes and fluid balances because it normally maintains about half of the extracellular fluids osmolarity.. Na+ is essential for the transmission of impulses in the nervous and muscle tissue.. Aldosterone stimulates the reabsorption of Na+... Decreased cardiac output and reduced blood volume are two triggers that cause the secretion of Aldosterone... A decline in Na+ blood concentration is the trigger mechanism for turning off ADH secretion... Absence of ADH enhances excretion of water in urine to restore Na+ to normal levels in the extracellular fluid (ECF)... The ANP is produced by the heart atria and is turned on when the Na+ levels rise to high and water has to be removed.. (The Internal Systems in a (GAS) Stress Response could have the Na+ and Aldosterone thresholds reset this would disrupt the ADH and the ANP ON-OFF Switch mechanisms).. Hypersecretion of aldosterone (Aldosteronism) can lead to Hypernatremia, and excessive water in the blood and decreased K+ (Potassium) blood levels.. When the water becomes excessive this raises the blood volume and causes high blood pressure.. The K+ depletion can become large enough to result in hyperpolarization of neurons and muscle fibers causing them to become desensitized.. Hypernataremia is excessive sodium blood levels... Sodium is the main regulator of the Osmotic Pressure of the ECF... Hypernataremia causes ECF Hypertonicity.. This leads to cell dehydration because water is drawn out of the body cells and goes into the ECF.. Some symptoms of this Cell Dehydration is intense thirst, restlessness, agitation, fatigue and high blood pressure.. (Hypertonicity combined with the lost Albumin due to the sugar concentrations effect on the permeability of the Endothelial-Capsular Membrane would cause a total disruption in the fluid compartments osmotic pressures.. Hypertonicity could be drawing water out of the Cells, while Albumin loss from the blood lowers Blood Colloid Osmotic Pressure allowing water to leave the blood, all that extra water in the Interstitial spaces would have to dehydrate something... And the excess water would have to go some where.)...

Another source of body water is the Metabolic water... Metabolic water is created mainly, in the Cellular Respiration Electron Transport Chain process when the Oxygen excepts and electron.. The Metabolic water volume hinges on the amount of ATPs being metabolized.. If the water loss is larger than the water increase this causes Dehydration.. (It seems to me that the excessive Aldosterone from a prolonged Stress Response coupled with the disrupted metabolism of ATPs due to the Nutrient Inhibitors inhibiting Iron causing an Oxygen deficiency, then add in the excessive Glucose drawing water out in the urine due to the disruption on the osmotic effect and it just stands to reason these could be triggering the Thirst Center)...

The Hunger /Satisfied Feeding Center :
First of all if the cell's are starving for nutrients then the Satisfied center will never be stimulated and the Hunger center will always remain activated... (It seems to me a person will be motivated by the Internal Systems to eat, eat, eat when the cells are on the verge of dying.)...

The following information traces God's Blueprints for FED and FASTING Phases of the Metabolic Process and is just another pathway that seems to confirm my theory, that it is Inhibited Nutrients that is the culprit behind the root cause of the Auto-immune disease epidemic..

Absorptive and Postabsorptive State of Metabolic Process:
FED State:
Metabolism is designed to keep a balance between the time when the blood contains a very rich concentration of Nutrients in the(Fed) Absorptive State and there is plenty ATP production being performed by the cells using Glucose as a fuel, and during the(Fasting) Postabsorptive State, the time when the body is forced to pull from its own reserves because nutrients are already used up in the GI tract . In the Fasting State substances normally stored-up, are instead, being torn down and mobilized to serve as fuel, because Glucose is not being absorbed through the GI tract.. (I believe, the Nutrient Inhibitors would lock the FASTING State in because the Glucose without the assistance of Oxygen would be unable to maintain a sufficient supply of ATPs... So all the Glucose would be designated for ATP metabolism and locked out of other metabolic processes... It would be as though no Glucose was available from the GI Tract.)... In the Fed State the processes that do the building-up exceed the processes that are doing the tearing-down and in the Fasting State the tearing-down processes exceeds the building-up processes... In the Fed State only a small amount of Amino acids and Fats are diverted to provide ATP, and the left over Amino Acids and Fats are being used to replenish the eroded resources of protein and fat, so they can be stored up for use during leaner times.. The hormone Insulin, almost single-handedly dominants the regulation in the Fed State... The trigger mechanism for Insulin secretion is the increase in blood Glucose levels.. The trigger mechanism for activating the Fasting State is depressed Insulin secretion..

(If the Nutrient Inhibitors disrupts ATP metabolism, the (GAS) "Fight or Flight" Stress Response would be to reset the normal threshold at which Glucose would be considered sufficient or deficient, thus changing the threshold for triggering Insulin release to a much higher level.. So even with an extremely high blood glucose level, if the metabolism process is caught-up in a the General Adaptation Syndrome "Fight or Flight" Stress Response, this would not trigger Insulin release if the "Tearing-Down" processes are still exceeding the "Building-Up" processes.. Amino Acid levels in the blood is a second Trigger Mechanism... (The Amino Acid Trigger Mechanism would be disrupted by low ATP diverting all the Glucose supply toward ATP production and away from any other metabolic processes, leaving no excess Glucose to be used in the other metabolic pathways thus disrupting the supply of Amino Acids)...

FASTING State:
Regulation of the Fasting State involves the Sympathetic Division of the Autonomic Nervous System and interaction with several hormones... The trigger mechanism for activating the Fasting State is depressed Insulin secretion.. The Glucagon Hormone is a yoked antagonist to Insulin... Glucgaon's purpose is to increase Glucose levels, so a decline in Glucose levels is the Trigger Mechanism that stimulates the secretion of Glucagon.. (So if the insufficient metabolism of ATPs activates the "Stress" Response from the Autonomic Nervous System's Sympathetic Division this opens the door for the General Adaptation Syndrome to reset the Glucose Threshold, thus requiring more Glucagon Hormone and suppressing Insulin... Even though there is a higher amount of Glucose in the blood, it would still register as too "low" to bring about ATP Homeostasis.. So as Insulin drops the cells insulin-induced responses decline as well... Since Homeostasis is disrupted by the Nutrient Inhibitors depletion of the resources, the Fasting State will remain activated till the supply is replenished.. And replenishing the resources is on a need to know bases, we need to know all the disruptions together so we can get a complete full picture, not just a single piece of the puzzle here and there.. Like Glucose is high and Insulin is low... What else is out of kilter? In my opinion this is why finding out what is in the Cell is so crucial to getting to a cure, and this is something I lay at the door of the medical community. In my opinion Nutrients Inhibitors are depleting the nutrient resource pool, this disrupts the flood of sufficient nutrient rich blood so that it can never arrive at the cells... And either the GAS Stress Response has reset the threshold causing the metabolism process to become desensitized to Insulin and/or the entrance into the Absorptive State is disrupted, in which case the Insulin is allowed to remain Switched-Off because its position as dominate hormone has not been activated and so all the metabolism functions Insulin is responsible for in the Metabolism Process are not being performed.)...

Summary:
There are two phases of the Metabolic Process: The Absorptive (FED)Phase and the Post absorptive (FASTING) Phase... These processes are responsible for maintaining a balanced nutrient concentration in the blood during times when nutrient are abundant and when nutrients are deficient.. In the FED Phase nutrients are fully supplied... In the FASTING Phase nutrients have to be obtained by breaking down the body's stores..(So in my opinion, if the Nutrient Inhibitors are interfering with the nutrient richness of the blood, causing the cells to become deprived of nutrients, this would keep the FED Phase Switched-Off automatically causing the Insulin to remain Switched-Off.. As the Insulin influence wanes from the metabolic process, then the FASTING hormones and neuronal controls become more dominant.. In my opinion the Fasting State has been permanently Switched-On and locked in by the Nutrient Inhibitors robbing the body of its ability to absorb nutrients... The cells' utilization of Calcium and Iron has been limited by the Insoluble Iron Complexes, as well as the Insoluble Calcium Salts, being created by the Nutrient Inhibitors.. This is altering the Electrolyte Concentration, creating an Imbalance that leads to a hostile environment that denatures functional proteins such as hemoglobin.. This destroys Hemoglobin's sites for carrying Oxygen, which would disrupt the Cellular Respiration Electron Transport Chain's metabolism of ATP... This would cause deficient ATPs which would make the body's Internal Systems react... Resulting in the following reactions from the Internal Systems. which could account for the metabolic disruptions seen in Diabetes Mellitus: Desensitized Insulin Receptors disrupting the cells ability to utilize Glucose, urine saturated with Glucose and high Glucose blood levels... Resulting in excessive destruction of fats and tissue protein so it can be utilized to produce energy.. Which can cause Metabolic Acidosis, protein wasting and weight loss.)...

Internal Responses to Starvation or Prolonged Fasting:
Starvation leads to a "Stress Shock Response".. Glucocorticoid secretion is increased.. Glycogen and Fat stores are depleted and tissue proteins become the source of blood Glucose.. Cellular amino acids are removed and converted to glucose (Gluconeogenesis) which depletes the cells resources for Amino Acids.. Then the Kidneys cells' Amino Acids are depleted to be converted to Glucose... Protein in the muscle tissue becomes the main target for getting blood Glucose, because moving is not as vital as the Immune System and healing wounds.. At a point, if the ATPs cannot be brought up to pare with normal adjustments of breaking down the tissue protein, then the internal response is to go into the Glucose Sparing Mode... Glucose Sparing has to kick-in because the heart is mainly muscle protein, and if the protein resources are depleted too much the heart will die.. In this Mode, the body beings to increase the utilization of non carbohydrate substances to burn more proteins and fats.. During prolonged Starvation the Brain manages to get its share of blood glucose, because all the other organs change to alternative energy resources, in the attempt to spare the Glucose for this most vital organ.. hGh is the hormone that influences this Glucose Sparing mechanism.. hGH plays an influential role in the metabolism of carbohydrate, protein and fat... It stops the uptake of Glucose by most cells making them switch away from Glucose to alternative sources for making ATPs... This frees up the Glucose for the Neurons' use.. If starvation continues Glucose Sparing makes the Adipose tissue release stored Fatty Acids so they can be oxidized by the tissue cells for energy.. The liver makes Ketone bodies by oxidizing fat, to supply the tissue cells with an alternative form of energy.. Eventually, when the Starvation gets to be too severe, the Brain will end up having to start utilizing large amounts of Ketone bodies to assist the Glucose as its energy fuel.. As the Brain switches over to this alternative energy fuel, this will slow down the tissue protein destruction and protect the heart muscle from destruction... But the damage of the Brain using an alternative fuel for a prolonged period of time has not been established.. (If the Nutrient Inhibitor induced "Cell Starvation" is not corrected this will only be a temporary fix.)..

06/22/2009

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